deep
peroneal nerve - Google Search
Fanconi
syndrome (also known as Fanconi's syndrome) is a disease of the proximal renal
tubules of the kidney in which glucose, amino acids, uric acid, phosphate and
bicarbonate are passed into the urine, instead of being reabsorbed.
Fanconi
syndrome (also known as Fanconi's syndrome) is a disease of the proximal renal
tubules[1] of the kidney in which glucose, amino acids, uric acid, phosphate
and bicarbonate are passed into the urine, instead of being reabsorbed. Fanconi
syndrome affects the proximal tubule, which is the first part of the tubule to
process fluid after it is filtered through the glomerulus. It may be inherited,
or caused by drugs or heavy metals.[2] lead po7soning
hypopyon -
Google Search
Myxedema or
myxoedema is a term used synonymously with severe hypothyroidism. It is also
used to describe a dermatological change that can occur in hypothyroidism, and
some forms of hyperthyroidism.
Hypertropia
is a condition of misalignment of the eyes (strabismus), whereby the visual axis
of one eye is higher than the fellow fixating eye. Hypotropia is the similar
condition, focus being on the eye with the visual axis lower than the fellow
fixating eye.
Fondaparinux
(trade name Arixtra) is an anticoagulant medication chemically related to low
molecular weight heparins
Oral glucose
Because GH
secretion is inhibited by glucose, measurement of glucose nonsuppressibility
may be useful, although there is debate as to what value of GH is considered
“normal” after a 1.75g/kg oral glucose challenge (not to exceed 75 g oral
glucose load). A paradoxic rise in GH concentration is seen in 15-20% of
patients with acromegaly following oral glucose administration.
Two baseline
GH levels are obtained prior to ingestion of 75 or 100 g of oral glucose, and
additional GH measurements are made at 30, 60, 90, and 120 minutes following
the oral glucose load.
With newer
assays for GH using the immunoradiometric assay (IRMA), acromegaly is thought
to be present when a criterion of less than 1 mcg/L is used following oral
glucose ingestion.
Growth
hormone
Patients
with active acromegaly have abnormal dynamics of GH secretion. A simple
diagnostic approach is to measure serum GH 1 hour after oral administration of
100 g of glucose. Clearly elevated GH levels (>10 ng/mL) after oral glucose,
combined with the clinical picture, secure the diagnosis of acromegaly, while a
normal GH level (< 5 ng/mL) after oral glucose essentially excludes the
diagnosis.
Only a small
percentage of patients investigated for acromegaly have a postglucose GH level
that is intermediate (5-10 ng/mL). In these patients, other tests can be used
to define their status.
Before
immunoassays for IGF-I, GH measurement was the only method used in the
biochemical assessment of acromegaly,[22] and the availability of
supersensitive GH has changed many aspects of the interpretation of the GH
value. Hypersecretion and abnormal neuroregulation characterize acromegaly. GH
can be measured in many ways to give useful information on diagnosis, therapy,
and prognosis. Measuring GH in the management of acromegaly complements the
information IGF-I values provide.
Random GH
measurements, however, often are not diagnostic, because of the episodic
secretion of GH, its short half-life, and the overlap between GH concentration
in individuals with acromegaly and individuals without the condition.
GHRH
GH-releasing
hormone (GHRH) concentration may be obtained if clinically indicated. levels
higher than 300 pg/mL usually indicate an ectopic source of GHRH. In pituitary
disease (GHRH independent), GHRH concentration is normal or suppressed.
Prolactin
Because as
many as 20% of GH-secreting pituitary adenomas co-secrete prolactin, the
prolactin level also may be elevated. However, a rise in prolactin may result
from stalk compression or from co-secretion from a pituitary adenoma. Pituitary
adenomas may be associated with deficiencies of other pituitary hormones.
Consider evaluation of the adrenal, thyroid, and gonadal axes.
Insulinlike
growth factor I
As
previously stated, IGF-I has been the most reliable biochemical indicator of
acromegaly. An excellent linear dose-response correlation between serum IGF-I
levels and 24-hour integrated GH secretion has been demonstrated. Elevated
IGF-I values in a patient whose symptoms prompt appropriate clinical suspicion
almost always indicate GH excess. IGF-I is useful not only in diagnosis, but
also in monitoring the efficacy of therapy.
Measurement
of IGF binding protein 3 (IGFBP-3), the primary binding protein for circulating
IGF, is increased in acromegaly and may be useful in its diagnosis. It also may
be helpful for monitoring the activity of the disease during treatment.
Factors
altering IGF-I levels
Starvation,
obesity, and diabetes mellitus decrease IGF-I concentration, while pregnancy
increases it. In addition, IGF-I concentrations vary with age, which means an
assay is required in which the normal ranges have been stratified to account
for this discrepancy.
Potential
confusion may arise in the evaluation of healthy adolescents, because IGF-I
levels can be substantially higher during puberty than during adulthood. Always
compare the patient's measurement with age-matched and sex-matched IGF-I
reference ranges published in the literature or established for the specific
testing laboratory.
Factors
altering IGF-I levels
Starvation,
obesity, and diabetes mellitus decrease IGF-I concentration, while pregnancy
increases i
heparin
induce thombocytopenia
Unlike other
forms of thrombocytopenia, HIT is generally not marked by bleeding; instead,
venous thromboembolism (eg, deep venous thrombosis, pulmonary embolism) is the
most common complication. Less often, arterial thrombosis (eg, myocardial
infarction) may occur. For that reason, the disorder is sometimes termed
heparin-induced thrombocytopenia and thrombosis (HITT).
ADHD is one
of the most common childhood disorders and can continue through adolescence and
into adulthood. The average age of onset is 7 years old.
The
association of postcricoid dysphagia, upper esophageal webs, and iron
deficiency anemia is known as Plummer-Vinson syndrome (PVS)
Histrionic
personality disorder (HPD) is defined by the American Psychiatric Association
as a personality disorder characterized by a pattern of excessive
attention-seeking emotions, usually beginning in early adulthood, including
inappropriately seductive behavior and an excessive need for approval.
secuctive
mean is tempting.. thayt mean atracting other peoples eye
Provocative
(or seductive) behavior
Relationships
are considered more intimate than they actually are
Attention-seeking
Influenced
easily
Speech
(style) wants to impress; lacks detail
Emotional
lability; shallowness
Make-up;
physical appearance is used to draw attention to self
Exaggerated
emotions; theatrical
SEDUCTIVE
BEHAVIOR OR HISTRIONIC PERSONALITY DISORDER
bush fire
pyromania
ˌpʌɪrə(ʊ)ˈmeɪnɪə/
noun
an obsessive
desire to set fire to things.
common cause
for bush fire
Pyromania is
an impulse control disorder[1] in which individuals repeatedly fail to resist
impulses to deliberately start fires,[1] in order to relieve tension or for
instant gratification.
Depersonalization
This article
is about the psychological symptom. For the diagnosis, see depersonalization
disorder.
Depersonalization
(or depersonalisation) is an anomaly of self-awareness. It can consist of a
reality or detachment within the self, regarding one's mind or body, or being a
detached observer of oneself. Subjects feel they have changed, and the world
has become vague, dreamlike, less real, or lacking in significance. It can be a
disturbing experience. Chronic depersonalization refers to
depersonalization/derealization disorder, which is classified by the DSM-5 as a
dissociative disorder.[1]
THAMAN
VENKENEK KIYALA HIYHAGENA EG. BARAC
OBAMA EEEVIDIYATA ACT KARANAVA.
DISSOCIATIVE
DISSODER AYATA ANIVARYEN DEPERSONALIAZATION THIYANAVA
Dissociative
disorders (DD) are conditions that involve disruptions or breakdowns of memory,
awareness, identity, or perception. People with dissociative disorders use
dissociation, a defense mechanism, pathologically and involuntarily.
Dissociative disorders are thought to primarily be caused by psychological
trauma.
Hypochondriasis,
or hypochondria, is an overwhelming fear that you have a serious disease, even
though health care providers can find no evidence of illness. People with
hypochondriasis misinterpret normal body sensations as signs of serious
illness.
Morbid
jealousy, also known as Othello syndrome or delusional jealousy, is a
psychological disorder in which a person is preoccupied with the thought that
their spouse or sexual partner is being unfaithful without having any real
proof, along with socially unacceptable or abnormal behaviour related to these
thoughts
cause for
pathological jealousy
Pathological
jealousy may be secondary to other psychiatric disorders, including
schizophrenia, depressive disorder, alcoholism, and organic disorder.
Pathological jealousy can also arise from a personality disorder, in
which the person has a pervasive sense of his own inadequacy, and a
vulnerability to anything that might
increase this sense of inadequacy, such as loss of status. In the face of such threats, the person may
project blame on to the partner, in the form of jealous accusations.
renin
angiotensin system - Google Search
symtopms
of ateract
Signs and
symptoms of cataracts include:
Clouded,
blurred or dim vision
Increasing
difficulty with vision at night
Sensitivity
to light and glare
Seeing
"halos" around lights
Frequent
changes in eyeglass or contact lens prescription
Fading or
yellowing of colors
Double
vision in a single eye
maculer
degenaration
macular
degeneration - Google Search
age related
maculer degenaration
macular
degeneration - Google Search
In current
medical practice, the renin-angiotensin-aldosterone-System's overactivity (and
resultant hypertension) is more commonly reduced using either ACE inhibitors
(such as ramipril and perindopril) or angiotensin II receptor blockers (ARBs,
such as losartan, irbesartan or candesartan) rather than a direct oral renin
inhibitor. ACE inhibitors or ARBs are also part of the standard treatment after
a heart attack.
Oral cholecystogram:
Abbreviated OCG. An x-ray procedure for diagnosing gallstones. The patient
takes iodine-containing tablets by mouth for one night or two nights in a row
extra hepatic billiary
obstrution following cholecystectomy
Biloma is a well recognized
postcholecystectomy complication that often accompanies biliary ductal injury.
ace inhibitors
Contraindications and
precautions
The ACE inhibitors are
contraindicated in patients with:
Previous angioedema
associated with ACE inhibitor therapy
Hypersensitivity to ACE
inhibitors
ACE inhibitors should be
used with caution in patients with:
Impaired renal function
Aortic valve stenosis or
cardiac outflow obstruction
Hypovolemia or dehydration
Hemodialysis with high-flux
polyacrylonitrile membranes
globus hystericus
ˌɡləʊbəs hɪˈstɛrɪkəs/
nounMEDICINE
the sensation of a lump in
the throat, as a symptom of anxiety or hysteria.
Globus pharyngis (also
known as globus sensation, globus or, somewhat outdatedly, globus hystericus,
commonly referred to as having a "lump in one's throat"), is the
persistent sensation of having phlegm, a pill or some other sort of obstruction
in the throat when there is none.
Schizophrenia is not
associated with any characteristic laboratory results.
Diagnostic criteria
According to the Diagnostic
and Statistical Manual of Mental Disorders, Fifth Edition, (DSM-5), to meet the
criteria for diagnosis of schizophrenia, the patient must have experienced at
least 2 of the following symptoms[1] :
Delusions
Hallucinations
Disorganized speech
Disorganized or catatonic
behavior
Negative symptoms
At least 1 of the symptoms
must be the presence of delusions, hallucinations, or disorganized speech.
Continuous signs of the
disturbance must persist for at least 6 months, during which the patient must
experience at least 1 month of active symptoms (or less if successfully
treated), with social or occupational deterioration problems occurring over a
significant amount of time. These problems must not be attributable to another
condition.
The American Psychiatric
Association (APA) removed schizophrenia subtypes from the DSM-5 because they
did not appear to be helpful for providing better-targeted treatment or
predicting treatment response.
renal stone
Stones that are 7 mm and
larger are unlikely to pass spontaneously and require some type of surgical
procedure, such as the following:
Stent placement
Percutaneous nephrostomy
Extracorporeal shockwave
lithotripsy
Ureteroscopy
Percutaneous
nephrostolithotomy
Open nephrostomy
See Treatment and
Medication for more detail.
Extracorporeal shockwave
lithotripsy
SWL, the least invasive of
the surgical methods of stone removal, utilizes high-energy sound waves focused
on the stone to shatter it into passable fragments. It is especially suitable
for stones that are smaller than 2 cm and lodged in the upper or middle calyx.
It is contraindicated in pregnancy, patients with untreatable bleeding
disorders, tightly impacted stones, or in cases of ureteral obstruction distal
to the stone. In addition, the effectiveness is limited for very hard stones
(which tend to be dense on CT scan), cystine stones, and in very large
patients.
phenytoin , carbamazaphine,
topiramidwith oral ctraceptive pilla
An interaction between
antiepileptic drugs (AEDs) and the combined oral contraceptive pill was first
proposed when the dose of estradiol in the oral contraceptive pill was reduced
from 100 to 50 microg. There was a higher incidence of breakthrough bleeding
and contraceptive failure among women with epilepsy compared with women in
general. Since then, interaction studies have been undertaken to look for
possible interactions between AEDs and the combined oral contraceptive pill.
Phenobarbital (phenobarbitone), phenytoin, carbamazepine, oxcarbazepine,
felbamate and topiramate have been shown to increase the metabolism of
ethinylestradiol and progestogens. Therefore, if a women is on one of the AEDs
and wishes to take the oral contraceptive pill, she will need to take a
preparation containing at least 50 microg of ethinylestradiol. Levonorgestrel
implants are contraindicated in women receiving these AEDs because of cases of
contraceptive failure. It is recommended that medroxyprogesterone injections be
given every 10 rather than 12 weeks to women who are receiving AEDs that induce
hepatic microsomal enzymes.
anancephally
Maternal serum
alpha-fetoprotein (MSAFP) screening during the second trimester of pregnancy is
an effective screening tool for identification of the vast majority of cases of
anencephaly in women with or without a positive family history or other risk
factors for neural tube defects.[18]
Amniotic alpha-fetoprotein
(AFAFP) testing during the late first trimester and second trimester of
pregnancy is a diagnostic biochemical test for anencephaly. False positives from
AFAFP can be excluded based on the results of testing for acetylcholinesterase
(ACHE), which should be clearly positive for open anencephaly.
su
Several types of hCG assay
have been proposed for use in prenatal screening. Second-trimester maternal
programs screening for T21 use either a beta-hCG assay or one that measures
free beta-subunit. In trials of first-trimester screening, free beta-subunit in
maternal serum and beta-core fragment in urine are under evaluation. The median
values for beta-hCG in trisomic pregnancies are higher than values in normal
pregnancies.
Schizophreniform disorder
is a short-term type of schizophrenia, a serious mental illness that distorts
the way a person thinks, acts, expresses emotions, perceives reality, and
relates to others. Like schizophrenia, schizophreniform disorder is a type of
"psychosis" in which a person cannot tell what is real from what is
imagined. Although schizophrenia is a lifelong illness, schizophreniform
disorder involves symptoms that are present for less than six months. When
symptoms persist longer than six months, the diagnosis is typically changed to
schizophrenia.
histrionic
ˌhɪstrɪˈɒnɪk/
adjective
1.
excessively theatrical or
dramatic in character or style.
About Borderline
Personality Disorder (BPD)
Brief Overview
Borderline personality
disorder (BPD) is a serious mental illness that causes unstable moods,
behavior, and relationships. It usually begins during adolescence or early
adulthood.
Most people who have BPD
suffer from:
Problems regulating their
emotions and thoughts
Impulsive and sometimes
reckless behavior
Unstable relationships
somatostatin and ocreotide(artficially
produce somatostatin analog)
Somatostatin,is a naturally
occurring tetradecapeptide isolated from the hypothalamus and from pancreatic
and enteric epithelial cells. (eg.gastric antram)
Through vasoconstriction,
somatostatin diminishes blood flow to the portal system, thus decreasing
variceal bleeding.(other than this somatostatin does some other funtion in the
body, it is inhibit GH .so we call GHIH)
It has effects similar to those of vasopressin
but does not cause coronary vasoconstriction.
Somatostatin has an initial
half-life of 1-3 minutes and is rapidly cleared from the circulation.
Somatostatin analogs
inhibit the secretion of hormones involved in vasodilation.
Octreotide is a synthetic
octapeptide.
Compared with somatostatin, octreotide has
similar pharmacologic actions with greater potency and longer duration of
action. In the US, octreotide is used off-label for the management of variceal
hemorrhage.[45]
Octreotide (Sandostatin)
and has a multitude of other endocrine and
nonendocrine effects, including the inhibition of glucagon, vasoactive
intestinal peptide, and GI peptides. Octreotide has greater potency and a
longer duration of action than somatostatin.
Beta-Blockers, Nonselective
Nonselective beta-blocking agents
decrease hepatic arterial and portal venous perfusion. Beta-adrenergic blockers
may block the effect of vasodilators, decrease platelet adhesiveness and
aggregation, and increase the release of oxygen to tissues.
Vasopressin, also known as
antidiuretic hormone (ADH), is a neurohypophysial hormone found in most
mammals. In most species it contains arginine and is thus also called arginine
vasopressin (AVP) or argipressin.[1] Its two primary functions are to retain
water in the body and to constrict blood vessels.[2] Vasopressin regulates the
body's retention of water
Pyoderma gangrenosum is an
uncommon, ulcerative cutaneous condition of uncertain etiology. It is
associated with systemic diseases in at least 50% of patients who are
affected.[1, 2] The diagnosis is made by excluding other causes of
similar-appearing cutaneous ulcerations, including infection, malignancy,
vasculitis, collagen vascular diseases, diabetes, and trauma.
abducens
nerve - Google Search
Postpartum Psychosis
Postpartum psychosis is the
most severe form of postpartum psychiatric illness.[58] The condition is rare,
occurring in approximately 1-2 per 1000 women after childbirth.[7] At highest
risk are women with a personal history of bipolar disorder or a previous episode
of postpartum psychosis.[59, 60]
Presentation
Postpartum psychosis has a
dramatic onset, emerging as early as the first 48-72 hours after delivery. In
most women, symptoms develop within the first 2 postpartum weeks.[58, 60] The
condition resembles a rapidly evolving manic or mixed episode, with symptoms
such as restlessness and insomnia, irritability, rapidly shifting depressed or
elated mood, and disorganized behavior.[58, 60]
The mother may have
delusional beliefs that relate to the infant (eg, the baby is defective or
dying, the infant is Satan or God), or she may have auditory hallucinations
that instruct her to harm herself or her infant.[3, 35, 43, 58, 60]
The risks for infanticide
and suicide are high among women with untreated postpartum psychosis.[35, 43]
Rates of infanticide in this population are as high as 4%.[43]
Management
Puerperal psychosis is a
psychiatric emergency that typically requires inpatient treatment.[20, 54, 60]
Most patients with postpartum psychosis have bipolar disorder. Acute treatment
includes a mood stabilizer (eg, lithium, valproic acid, carbamazepine) in
combination with antipsychotic medications and benzodiazepines.[20]
Electroconvulsive therapy
(ECT) (often bilateral) is well tolerated and rapidly effective.[53]
Breastfeeding and
Psychotropic Medications
Women who plan to
breastfeed must be informed that all psychotropic medications, including
antidepressants, are secreted into breast milk. Concentrations in breast milk
vary widely.[61] Infant serum blood levels of antidepressants are not typically
obtained unless the question of toxicity in the infant arises.
Data on the use of
tricyclic antidepressants, fluoxetine, sertraline, and paroxetine during
breastfeeding are encouraging, and serum antidepressant levels in the breastfed
infant are either low or undetectable. Reports of toxicity in breastfed infants
are rare, although the long-term effects of exposure to trace amounts of
medication are not known.[4]
Women treated with valproic
acid and carbamazepine should avoid breastfeeding, because these agents have
been associated with hepatotoxicity in the infant.[4] In addition, avoid
breastfeeding in premature infants or in those with hepatic insufficiency who
may have difficulty metabolizing medications present in breast milk.[4]
Breastfeeding in women
treated with lithium should be pursued with caution because lithium is secreted
at high levels in breast milk and may cause significant toxicity in the nursing
infant. If the breastfed infant is exposed to lithium in the breast milk,
periodic monitoring of lithium levels and thyroid function is indicated.[4, 61]
Women with bipolar disorder
or a history of postpartum psychosis may benefit from prophylactic treatment
with lithium, initiated either before or within 24 hours of delivery.
Stages of Alzheimer's
disease[18]
Effects of ageing on memory
but not AD
Forgetting things
occasionally
Misplacing items sometimes
Minor short-term memory
loss
Not remembering exact
details
Early stage Alzheimer's
Not remembering episodes of
forgetfulness
Forgets names of family or
friends
Changes may only be noticed
by close friends or relatives
Some confusion in
situations outside the familiar
Middle stage Alzheimer's
Greater difficulty
remembering recently learned information
Deepening confusion in many
circumstances
Problems with sleep
Trouble knowing where they
are
Late stage Alzheimer's
Poor ability to think
Problems speaking
Repeats same conversations
More abusive, anxious, or
paranoid
Alzheimer disease (AD) is
an acquired disorder of cognitive and behavioral impairment that markedly
interferes with social and occupational functioning. It is an incurable disease
with a long and progressive course. In AD, plaques develop in the hippocampus,
a structure deep in the brain that helps to encode memories, and in other areas
of the cerebral cortex that are used in thinking and making decisions. Whether
plaques themselves cause AD or whether they are a by-product of the AD process
is still unknown. See the image below.
Coronal T1-weighted
magnetic resonance imaging (MRI) scan in a patient with moderate Alzheimer
disease. Brain image reveals hippocampal atrophy, especially on the right side.
View Media Gallery
Signs and symptoms
Preclinical Alzheimer
disease
A patient with preclinical
AD may appear completely normal on physical examination and mental status
testing. Specific regions of the brain (eg, entorhinal cortex, hippocampus)
probably begin to be affected 10-20 years before any visible symptoms appear.
Mild Alzheimer disease
Signs of mild AD can
include the following:
Memory loss
Confusion about the
location of familiar places
Taking longer to accomplish
normal, daily tasks
Trouble handling money and
paying bills
Compromised judgment, often
leading to bad decisions
Loss of spontaneity and
sense of initiative
Mood and personality
changes; increased anxiety
Moderate Alzheimer disease
The symptoms of this stage
can include the following:
Increasing memory loss and
confusion
Shortened attention span
Problems recognizing
friends and family members
Difficulty with language;
problems with reading, writing, working with numbers
Difficulty organizing
thoughts and thinking logically
Inability to learn new
things or to cope with new or unexpected situations
Restlessness, agitation,
anxiety, tearfulness, wandering, especially in the late afternoon or at night
Repetitive statements or
movement; occasional muscle twitches
Hallucinations, delusions,
suspiciousness or paranoia, irritability
Loss of impulse control:
Shown through behavior such as undressing at inappropriate times or places or
vulgar language
Perceptual-motor problems:
Such as trouble getting out of a chair or setting the table
Severe Alzheimer disease
Patients with severe AD
cannot recognize family or loved ones and cannot communicate in any way. They
are completely dependent on others for care, and all sense of self seems to
vanish.
Other symptoms of severe AD
can include the following:
Weight loss
Seizures, skin infections,
difficulty swallowing
Groaning, moaning, or
grunting
Increased sleeping
Lack of bladder and bowel
control
In end-stage AD, patients
may be in bed much or all of the time. Death is often the result of other
illnesses, frequently aspiration pneumonia.
See Clinical Presentation for
more detail.
Endogenous Depression is an
atypical sub-class of the mood disorder, major depressive disorder (clinical
depression). Endogenous depression includes patients with treatment-resistant,
non-psychotic, major depressive disorder, characterized by abnormal behavior of
the endogenous opioid system but not the monoaminergic system.
Endogenous depression is a
type of major depressive disorder (MDD). Although it used to be seen as a
distinct disorder, endogenous depression is now rarely diagnosed. Instead, it’s
currently diagnosed as MDD. MDD, also known as clinical depression, is a mood
disorder characterized by persistent and intense feelings of sadness for
extended periods of time. These feelings have a negative impact on mood and
behavior as well as various physical functions, including sleep and appetite.
How Does Endogenous
Depression Differ from Exogenous Depression?
Researchers used to
differentiate endogenous depression and exogenous depression by the presence or
absence of a stressful event before the onset of MDD:
Alcoholic hallucinosis vs.
delirium tremens
Both alcoholic hallucinosis
and DTs have been thought of as different manifestations of the same
physiological process in the body during alcohol withdrawal.[8]Alcoholic hallucinosis
is a much less serious diagnosis than delirium tremens. Delirium tremens (DTs)
do not appear suddenly, unlike alcoholic hallucinosis. DTs also take
approximately 48 to 72 hours to appear after the heavy drinking stops. A tremor
develops in the hands and can also affect the head and body. A common symptom
of delirium tremens is that people become severely uncoordinated. The biggest
difference between alcoholic hallucinosis and delirium tremens is that
alcoholic hallucinosis have a much better prognosis than DTs.[9] Moreover,
delirium tremens can be fatal when untreated.[5]
treat for alcoholic
hallucination
Monday, February
22, 2016
9:43 AM
Alcohol abuse can create a
deficiency of thiamine, magnesium, zinc, folate and phosphate as well as cause
low blood sugar.[10] However, several tested drugs have shown the disappearance
of hallucinations. Neuroleptics and benzodiazepines showed normalization.
Common benzodiazepines are chlordiazepoxide and lorazepam. It’s been shown that
management has been effective with a combination of abstinence from alcohol and
the use of neuroleptics.[1
alcoholic hallocination
mainly due to auditory..
Alcoholic hallucinosis is a rare
condition characterized by distressing auditory hallucinations (usually
of voices uttering threats) occurring in clear consciousness. Some patients
argue aloud with the voices, others feel compelled to follow instructions from
them. Delusional misinterpretations may follow, often of a persecutory kind, so
that the clinical picture can resemble
schizophrenia. The condition can arise while the person is still drinking
heavily, or when intake has been reduced. It
is of variable duration and when chronic needs to be distinguished from
a primary schizophrenic disorder. It
usually responds to antipsychotics and has a good prognosis. The
symptoms may reoccur if drinking resumes.
Lung cancers are generally
divided into two main categories: small cell lung cancer (SCLC) and non–small
cell lung cancer (NSCLC). NSCLC accounts for approximately 85% of all lung
cancers. Histologically, NSCLC is divided further into adenocarcinoma, squamous
cell carcinoma (SCC), and large cell carcinoma. (See Pathophysiology.)
cancer is known to be
caused by tobacco smoking in as many as 90% of patients. However, two recent
studies have reported rising NSCLC rates in persons who have never smoked:
small cell lung cancer are
usually due to smaking
adeno carcinoma are now
becoming more than squaremus cal CA. amost of adeno ca due to smarning.. but
non smaker also get adeno ca
chart of
lung cancers.svg - Wikipedia, the free encyclopedia
cause for gunecomastia
Antiandrogen/inhibitors of
androgen synthesis are as follows:
Cyproterone acetate
Flutamide, bicalutamide,
nilutamide
Finasteride, dutasteride
Ketoconazole
Spironolactone
Tea tree oil
Cancer/chemotherapeutic
drugs are as follows:
Alkylating agents
Methotrexate
Vinca alkaloids
Imatinib
Cardiac and
antihypertensive medications are as follows:
Calcium channel blockers
(verapamil, nifedipine, diltiazem)
ACE Inhibitors (captopril,
enalapril)
Digoxin
Alpha-blockers
Amiodarone
Methyldopa
Reserpine
Nitrates
Hormones are as follows:
Androgens
Anabolic steroids
Estrogens
Growth hormones
Chorionic gonadotropin
Psychoactive drugs are as
follows:
Haloperidol
Diazepam
Tricyclic antidepressants
Haloperidol
Phenothiazines
Drugs for infectious
diseases are as follows:
Antiretroviral therapy for
HIV/AIDS (eg, indinavir)
Isoniazid
Ethionamide
Griseofulvin
Minocycline
Metronidazole
Ketoconazole
Drugs of abuse are as
follows:
Amphetamines
Heroin
Methadone
Alcohol
Marijuana
Others are as follows:
Theophylline
Omeprazole
Auranofin
Diethylpropion
Domperidone
Penicillamine
Sulindac
Heparin
Methotrexate
Two common presentations of
patients with IgA nephropathy are episodic gross hematuria and persistent
microscopic hematuria. Recurrent macroscopic hematuria, usually associated with
an upper respiratory tract infection, or, less often, gastroenteritis is the
most frequent clinical presentation and is observed in 40-50% of presenting
patients. In 30-40% of patients, the disease is asymptomatic, with erythrocytes
(RBCs), RBC casts, and proteinuria discovered on urinalysis. Patients with IgA
nephropathy can also present with acute kidney injury or chronic kidney disease
The importance of the
therapeutic relationship, also known as therapeutic alliance, between client
and therapist is often regarded as crucial to psychothera
What is psychoanalytic
psychotherapy?
What is psychoanalytic or
psychodynamic psychotherapy?
Psychoanalytic or
psychodynamic psychotherapy draws on theories and practices of analytical
psychology and psychoanalysis. It is a therapeutic process which helps patients
understand and resolve their problems by increasing awareness of their inner
world and its influence over relationships both past and present. It differs
from most other therapies in aiming for deep seated change in personality and
emotional development.
Psychoanalytic and
psychodynamic psychotherapy aim to help people with serious psychological
disorders to understand and change complex, deep-seated and often unconsciously
based emotional and relationship problems thereby reducing symptoms and
alleviating distress.
Sometimes people seek help
for specific reasons such as eating disorders, psycho-somatic conditions,
obsessional behaviour, or phobic anxieties. At other times help is sought
because of more general underlying feelings of depression or anxiety,
difficulties in concentrating, dissatisfaction in work or inability to form satisfactory
relationships.
The Billings Ovulation
Method (BOM) is a method which women use to monitor their fertility, by
identifying when they are fertile and when they are infertile during each
ovarian/menstrual cycle. Users pay attention to the sensation at their vulva,
and the appearance of any vaginal discharge
billing methord of
contraception
Monday, February
22, 2016
11:11 AM
To use the cervical mucus
method, it's important to understand how cervical secretions change during a
typical menstrual cycle. Generally, you'll have:
No noticeable cervical
secretions for three to four days after your period ends
Scanty, cloudy and sticky
secretions for the next three to five days
Abundant, clear and wet
secretions for the next three to four days — the period before and during
ovulation
No noticeable cervical
secretions for 11 to 14 days until your next period begins
). Patients with ALL can
have a high, normal, or low white blood cell (WBC) count, but they usually
exhibit neutropenia
One third of patients who
experience STEMI die within 24 hours of the onset of ischemia, and many of the
survivors experience significant morbidity.
Acute myocardial infarction
is associated with a 30% mortality rate; half of the deaths occur prior to
arrival at the hospital. An additional 5-10% of survivors die within the first
year after their myocardial infarction. Approximately half of all patients with
a myocardial infarction are rehospitalized within 1 year of their index event.
Levodopa-induced dyskinesia
is a form of dyskinesia associated with levodopa used to treat Parkinson's
disease. It often involves hyperkinetic movements, including chorea, dystonia,
and athetosis.[1]
rectal cancer
Bleeding is the most common
symptom of rectal cancer, occurring in 60% of patients.
Change in bowel habits is
present in 43% of patients; change is not evident in some cases because the
capacity of a rectal reservoir can mask the presence of small lesions. When
change does occur it is often in the form of diarrhea, particularly if the
tumor has a large villous component.
Unlike obstructive lung
diseases, including asthma and COPD, which show a normal or increased total
lung capacity (TLC), restrictive disease are associated with a decreased TLC.
Congenital rubella syndrome
(CRS) can occur in a developing fetus of a pregnant woman who has contracted
rubella, usually in the first trimester. If infection occurs 0–28 days before
conception, the infant has a 43% chance of being affected. If the infection
occurs 0–12 weeks after conception, the chance increases to 51%. If the
infection occurs 13–26 weeks after conception, the chance is 23% of the infant
being affected by the disease. Infants are not generally affected if rubella is
contracted during the third trimester, or 26–40 weeks after conception.
Common causes of postcoital
bleeding include: cervical pathology (dysplasia), sexually transmitted
infections (chlamydia or gonorrhea), or cervical polyps. Vaginitis, caused by
an infection such as Trichomonas or Candida, can also result in bleeding but
the patient would likely be symptomatic, complaining of discharge, itching, or
irritation
Hot nodules are virtually
never malignant. If the nodule is cold, chances are still good that it’s not
malignant – but not quite as good: about 10% of cold nodules turn out to be
malignant.
Mycosis fungoides, also
known as Alibert-Bazin syndrome or granuloma fungoides, is the most common form
of cutaneous T-cell lymphoma. It generally affects the skin, but may progress
internally over time. Symptoms include rash, tumors, skin lesions, and itchy
skin.
supiurative hidroadenitis
Hidradenitis suppurativa is
a disorder of the terminal follicular epithelium in the apocrine gland–bearing
skin. This condition is a chronic disabling disorder that relentlessly
progresses, frequently causing keloids, contractures, and immobility. See the
image below
dermatogibroma
Dermatofibroma (superficial
benign fibrous histiocytoma) is a common cutaneous nodule of unknown etiology
that occurs more often in women. Dermatofibroma frequently develops on the
extremities (mostly the lower legs) and is usually asymptomatic, although
pruritus and tenderness can be present.
Parasympathetic tone from
these sources are obviously well matched to sympathetic tone in healthy people.
Hyperstimulation of parasympathetic influence promotes bradyarrhythmias. When
hyperstimulated, the left vagal branch predisposes the heart to conduction
block at the atrioventricular node.
the substance released by
the vagus nerve as vagusstoff, which was later found to be acetylcholine. Drugs
that inhibit the muscarinic receptors (anticholinergics) such as atropine and
scopolamine, are called vagolytic because they inhibit the action of the vagus
nerve on the heart, gastrointestinal tract, and other organs. Anticholinergic
drugs increase heart rate and are used to treat bradycardia.
addison's
disease - Google Search
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